Citation: | SHI Min, WANG Yugang, WANG Ting, WANG Na, WEI Jue, CAI Zihao. Role of NOX1 and NOX3 in mice with Non-alcoholic steatohepatitis[J]. Journal of Clinical Medicine in Practice, 2012, (23): 5-8. |
Duvnjak M, Lerotic I, Barsic N. Pathogenesis and management issues for non-alcoholic fatty liver disease [J]. World Journal of Gastroenterology, 2007, (34):4539.
|
Chang T, Wang R, Wu L. Methylglyoxal-induced nitric oxide and peroxynitrite production in vascular smooth muscle cells [J]. Free Radical Biology and Medicine, 2005.286.
|
Leiter L A, Lewanczuk R Z. Of the renin-angiotensin system and reactive oxygen species Type 2 diabetes and angiotensin Ⅱ inhibition [J]. American Journal of Hypertension, 2005.121.
|
Kawahara T, Quinn M T, Lambeth J D. Molecular evolution of the reactive oxygen-generating NADPH oxidase (Nox/Duox)family of enzymes [J]. Bmc Evolutionary Biology, 2007.109.
|
Vignais P V. The superoxide-generating NADPH oxidase:structural aspects and activation mechanism [J]. Cellular and Molecular Life Sciences, 2002.1428.
|
Bedard K, Krause K H. The NOX family of ROS-generating NADPH oxidases:physiology and pathophysiology [J]. Physiological Reviews, 2007.245.
|
Kallwitz E R, Mclachlan A, Cotler S J. Role of peroxisome proliferators-activated receptors in the pathogenesis and treatment of nonalcoholic fatty liver disease [J]. World Journal of Gastroenterology, 2008(1):22.doi: 10.3748/wjg.14.22.
|
Adams L A, Angulo P, Lindor K D. Nonalcoholic fatty liver disease [J]. Canadian Medical Association Journal, 2005(7):899.
|
Chitturi S, Farrell G C. Etiopathogenesis of nonalcoholic steatohepatitis [J]. Seminars in Liver Disease, 2001.27.
|
Csak T, Ganz M, Pespisa J. Fatty acid and endotoxin activate inflammasomes in mouse hepatocytes that release danger signals to stimulate immune cells [J]. Hepatology, 2011.133.
|
Song F, Jia W, Yao Y. Oxidative stress, antioxidant status and DNA damage in patients with impaired glucose regulation and newly diagnosed Type 2 diabetes [J]. Clinical Science(London), 2007.599.
|
Kaneto H, Katakami N, Kawamori D. Involvement of oxidative stress in the pathogenesis of diabetes [J]. Antioxidants and Redox Signalling, 2007.355.
|
Kowaltowski A J, de Souza-Pinto N C, Castilho R F. Mitochondria and reactive oxygen species [J]. Free Radical Biology and Medicine, 2009(4):333.
|
Bedard K, Krause K H. The NOX family of ROS-generating NADPH oxidases;physiology and pathophysiology [J]. Physiological Reviews, 2007(1):245.
|
Bartosz G. Reactive oxygen species;destroyers or messengers [J]. Biochemical Pharmacology, 2009(8):1303.
|
Pendyala S, Natarajan V. Redox regulation of Nox proteins [J]. Respiratory Physiology & Neurobiology, 2010(3):265.
|
Pricci F. Oxidative stress in diabetes-induced endothelial dysfunction involvement of nitric oxide and protein kinase C [J]. Free Radical Biology and Medicine, 2003.683.
|
Park H S, Chun JN, Jung H Y. Hole ofNADPH oxidnse 4 in lipopolysncchnride-induced proinflammatory responses by human aortic endothelial cells [J]. Cardinvas Res, 2006.447.
|